Impaired tolerance to nutritional fats and its contribution to atherothrombogenesis


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M G Bubnova

R G Oganov

References

  1. Zilversmit D. В. Atherogenesis: a postprandial phenomenon. Circulation 1919, 60: 473-484.
  2. Mamo J. С. L., Proctor S. В., Smith D. Retention of chylomicron remnants by arterial tissue; importance of an efficient clearance mechanism from plasma. Atherosclerosis 1998; 141 (suppl. 1): S63-S69.
  3. Gotto A. M., Patsch J., Yamamoto A. Postprandial hyperlipidemia. Am. J. Cardiol. 1991; 68: 11-12.
  4. Erlcelens D. W. Man is a postprandial subject and postprandial triglyceride-rich lipoprotein remnants are atherogenic. Nutr. Metab. Cardiovac. Dis. 1993; 3: 99-101.
  5. Krauss R. M., Williams P. Т., Brensike J. et al. Intermediatedensity lipoproteins and progression of coronary artery disease in hypercholesterolemic men. Lancet. 1987; 2: 62-66.
  6. Mahley R. W., Hussain M. M. Chylomicron and chylomicron remnant catabolism. Curr. Opin. Lipidol. 1991; 2: 170-176.
  7. Mann С. J., Yen F. Т., Grant A. M., Bihain В. Е. Mechanism of plasma cholesteryl ester transfer in hypertriglyceridemia. J. Clin. Invest. 1991; 88: 2059-2066.
  8. Mamo J. С. L. Atherosclerosis as a postprandial disease. Endocr. Metab. 1995; 229-244.
  9. Mamo J. С. L., Yu К. С. W., Elsegood С. L. et al. Is atherosclerosis exclusively a post-prandial phenomenon? Clin. Exp. Pharmacol. Physiol. 1997; 24: 288-293.
  10. Zilversmit D. B. Mechamisms of cholesterol accumulation in the arterial wall. Am. J. Cardiol. 1975; 35: 559.
  11. Ross A. C., Zilversmit D. B. Chylomicron remnant cholesteryl esters as the major constituent of very low density lipoproteins in plasma of cholesterol-fed rabbits. J. Lipid Res. 1977; 18: 169.
  12. Corey J. E., Zilversmit D. В. Effect of cholesterol feeding on arterial lipolytic activity in the rabbit. Atherosclerosis 1977; 27: 201.
  13. Cabin H. C., Schwartz D. E., Virmani R. et al. Type III hyperlipoproteinemia: quantification, distribution, and nature of atherosclerotic coronary arterial narrowing in five necropsy patients. Am. Heart J. 1981; 102: 830-835.
  14. Mobile L., Salvayre R., Bonnafe M. J., Negre-Salnayre A. Oxidizability and subsequent cytotoxicity of chylomicron to monocytic U 937 and endothelial cells are dependent on dietary fatty acid composition. Biol, and Med. 1995; 5: 599-607.
  15. Patsch J. R., Miesenbock G., Hopferwieser T. et al. Relation of triglyceride metabolism and coronary artery disease: studies in the postprandial state. Arterioscler. Thromb 1992; 12: 1336- 1345.
  16. Davignon J., Conn J. Triglycerides: a risk factor for coronary heart disease (Review). Atherosclerosis 1996; 124 (suppl. 1): S57-S64.
  17. International Committee for the evaluation of hypertriglyceridemia as a vascular risk factor. Hypertriglyceridemias: risk and management. G. Lorenzini Med. Found. Milan, Italy, Houston, USA. 1-20.
  18. Patsch J. R., Karlin J. В., Scolt L. W. et al. Inverse relationship between levels of high density lipoprotein subfraction 2 and magnitude of postprandial lipemia. Proc. Natl. Acad. Sci. USA, 1983; 80: 1449-1453.
  19. Parkard C. J., Shepherd J. Lipoprotein heterogeneity and apolipoprotein В metabolism. Arterioscler. Thromb. Vase. Biol. 1997; 17: 3543-3556.
  20. Wu A. L., Windmueler H. G. Relative contributions by liver and intestine to individual plasma apolipoproteins in the rat. J. Biol. Chem. 1979; 254: 7316-7322.
  21. Eisenberg S. Relationship of HDL to other lipoproteins. In: Gotto A. M. et al., eds. High density lipoproteins and atherosclerosis. Amsterdam: Elsevier Press: 1978. 67-75.
  22. Patsch J. R., Prasad S., Gotto A. M., Bengtsson-Olivecrona G. Postprandial lipemia. A key for the conversion of high density lipoprotein 2 into high density lipoprotein 3 by hepatic lipase. J. Clin. Invest. 1984; 74: 2017-2023.
  23. Chan L., Dresel H. A. Biology of disease. Genetic factors in fluencing lipoprotein structure: implications for atherosclerosis. Lab. Invest. 1990; 62: 522-537.
  24. Tall A. R. Plasma high density lipoproteins. Metabolism and relationship to atherogenesis. J. Clin. Invest. 1990; 86: 379- 384.
  25. Власов В. В. Введение в доказательную медицину. Москва: Медиа Сфера; 2001. 177-180.
  26. Gronholt M. L. M., Nordestgaard В. G., Wiebe В. М. et al. Echo-lucency of computerized ultrasound images of carotid atherosclerotic plaques are associated with increased levels of triglvceride-rich lipoproteins as well as increased plaque lipid content. Circulation 1998; 97: 34-40.
  27. Havel R. J. Postprandial hyperlipidemia and remnant lipoproteins. Curr. Opin. Lipidol. 1994; 5: 102-109.
  28. Patsch J. R., Hopferwieser T., Muhlberger V. et al. Postprandial lipemia in patients with coronary artery disease. Circulation 1990; 82 (suppl. Ill): S284.
  29. Steiner G. Triglyceride-rich lipoproteins and atherosclerosis, from fast to feast. Ann. Med. 1993; 25: 431-435.
  30. Weintraub M. S., Grosskopf I., Rassin T. et al. Clearance of chylomicron remnants in normolipidaemic patients with coronary arterv disease: case control study over three years. Br. Med. J. 1996; 312: 936-939.
  31. Slampfer M. J., Krauss R. M., Blanche P. J. et al. A prospective study of triglyceride level, low-density lipoprotein particle diameter, and risk of myocardial infarction. J. A. M. A. 1996; 276: 882-888.
  32. Kugigyama K., Doi H., Takazol K. et al. Remnant lipoprotein levels in fasting serum predict coronary events in patients with coronary artery disease. Circulation 1999; 99: 2858-2860.
  33. Karpe F., Steiner G., Uffelman K. et al. Postprandial lipoproteins and progression of coronary atherosclerosis. Atherosclerosis 1994; 106: 83-97.
  34. Perova N. V., Metelskaya V. A., Bubnova M. G. et. al. Association of apo E polymorphism with nutritional and drug-induced changes in lipoproteins. Eur. J. Lab. Med. 1997; 5: 1-5.
  35. Kesaniemi Y. A., Ehnolm C., Miettinen T. A. Intestinal cholesterol absorption efficiency in man is related to apoprotein E phenotype. J. Clin. Invest. 1987; 80: 578-581.
  36. Соколов Е. И., Старкова Н. Т., Щукина Г. Н. и др. Метаболический синдром X как основа ишемической болезни сердца. Кардиология 1997; 3: 4-7.
  37. Аронов Д. М., Бубнова М. Г., Перова Н. В. и др. Влияние максимальной и субмаксимальной физических нагрузок на алиментарную дислипидемию. Тер. арх. 1993; 3: 57-62.
  38. Перова Н. В., Бубнова М. Г., Аронов Д. М. и др. Влияние ловастатина на концентрацию липидов и аполипопротеинов сыворотки крови после алиментарной жировой нагрузки у больных ишемической болезнью сердца. Кардиология 1993; 4: 8-10.
  39. Бубнова М. Г., Аронов Д. М., Перова Н. В. и др. Правастатин в коррекции атерогенной экзогенно-индуцированной постпрандиальной гиперлипидемии. Там же 2002; 1: 27- 32.
  40. Бубнова М. Г., Аронов Д. М., Перова Н. В. и др. Никотиновая кислота (пролонгированная форма эндурацин) повышает толерантность липид-транспортной системы к атерогенному воздействию пищевых жиров. Клин, фармакол. и тер. 2002; 11(4): 26-30.

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