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<article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance" xmlns:ali="http://www.niso.org/schemas/ali/1.0/" article-type="review-article" dtd-version="1.2" xml:lang="en"><front><journal-meta><journal-id journal-id-type="publisher-id">Terapevticheskii arkhiv</journal-id><journal-title-group><journal-title xml:lang="en">Terapevticheskii arkhiv</journal-title><trans-title-group xml:lang="ru"><trans-title>Терапевтический архив</trans-title></trans-title-group></journal-title-group><issn publication-format="print">0040-3660</issn><issn publication-format="electronic">2309-5342</issn><publisher><publisher-name xml:lang="en">LLC Obyedinennaya Redaktsiya</publisher-name></publisher></journal-meta><article-meta><article-id pub-id-type="publisher-id">31276</article-id><article-categories><subj-group subj-group-type="toc-heading" xml:lang="en"><subject>Editorial article</subject></subj-group><subj-group subj-group-type="toc-heading" xml:lang="ru"><subject>Передовая статья</subject></subj-group><subj-group subj-group-type="article-type"><subject>Review Article</subject></subj-group></article-categories><title-group><article-title xml:lang="en">Anemia and gastrointestinal tract diseases</article-title><trans-title-group xml:lang="ru"><trans-title>Анемия и заболевания желудочно-кишечного тракта</trans-title></trans-title-group></title-group><contrib-group><contrib contrib-type="author"><name-alternatives><name xml:lang="en"><surname>Luzina</surname><given-names>E V</given-names></name><name xml:lang="ru"><surname>Лузина</surname><given-names>Е В</given-names></name></name-alternatives><email>el.luz@list.ru</email><xref ref-type="aff" rid="aff1"/></contrib><contrib contrib-type="author"><name-alternatives><name xml:lang="en"><surname>Lareva</surname><given-names>N V</given-names></name><name xml:lang="ru"><surname>Ларева</surname><given-names>Н В</given-names></name></name-alternatives><xref ref-type="aff" rid="aff1"/></contrib></contrib-group><aff-alternatives id="aff1"><aff><institution xml:lang="en"></institution></aff><aff><institution xml:lang="ru">Читинская государственная медицинская академия</institution></aff></aff-alternatives><pub-date date-type="pub" iso-8601-date="2013-04-15" publication-format="electronic"><day>15</day><month>04</month><year>2013</year></pub-date><volume>85</volume><issue>4</issue><issue-title xml:lang="en">VOL 85, NO4 ()</issue-title><issue-title xml:lang="ru">ТОМ 85, №4 (2013)</issue-title><fpage>102</fpage><lpage>105</lpage><history><date date-type="received" iso-8601-date="2020-04-10"><day>10</day><month>04</month><year>2020</year></date></history><permissions><copyright-statement xml:lang="en">Copyright ©; 2013, Consilium Medicum</copyright-statement><copyright-statement xml:lang="ru">Copyright ©; 2013, ООО "Консилиум Медикум"</copyright-statement><copyright-year>2013</copyright-year><copyright-holder xml:lang="en">Consilium Medicum</copyright-holder><copyright-holder xml:lang="ru">ООО "Консилиум Медикум"</copyright-holder><ali:free_to_read xmlns:ali="http://www.niso.org/schemas/ali/1.0/"/><license><ali:license_ref xmlns:ali="http://www.niso.org/schemas/ali/1.0/">https://creativecommons.org/licenses/by-nc-sa/4.0</ali:license_ref></license></permissions><self-uri xlink:href="https://ter-arkhiv.ru/0040-3660/article/view/31276">https://ter-arkhiv.ru/0040-3660/article/view/31276</self-uri><abstract xml:lang="en"><p>The paper considers the causes of iron- and cyanobalamin-deficiency anemias caused by gastrointestinal tract (GT) diseases, among which malabsorption along with loss of these nutrients through the GT is of great importance. The paper reflects the current views of the pathogenesis of deficiency anemias that develop in gluten-sensitive celiac disease and atrophic gastritis. Among the atrophic gastritides, there are two forms caused by autoimmune processes and long-term Helicobacter pylori persistence, whose treatment is an effective measure in refractory anemia. The paper gives the provisions of the Russian Gastroenterology Association Guidelines (2012) for the management of H. pylori infection, which are based on Maastricht IV consensus (2010).</p></abstract><trans-abstract xml:lang="ru"><p>Аннотация. В статье рассматриваются причины развития железо- и цианокобаламиндефицитных анемий, обусловленных заболева­ниями желудочно-кишечного тракта (ЖКТ), среди которых наряду с потерей этих нутриентов через ЖКТ большое значение имеет нарушение всасывания. Статья отражает современные взгляды на патогенез дефицитных анемий, формирующихся при чувствительной к глютену целиакии и атрофическом гастрите. Среди атрофических гастритов выделяют 2 формы, формирование которых происходит за счет аутоиммунных процессов, а также под воздействием длительного персистирования Helicobacter pylori, лечение которой является эффективным мероприятием при рефрактерной анемии. В статье приводятся положения Рекомендаций Российской гастроэнтерологической ассоциации (2012) по лечению инфекции H. pylori, опирающиеся на Маастрихтское соглашение IV (2010).</p></trans-abstract><kwd-group xml:lang="en"><kwd>anemia</kwd><kwd>gastrointestinal tract</kwd><kwd>gluten-sensitive celiac disease</kwd><kwd>autoimmune gastritis</kwd><kwd>Helicobacter pylori infectio</kwd></kwd-group><kwd-group xml:lang="ru"><kwd>анемия</kwd><kwd>желудочно-кишечный тракт</kwd><kwd>чувствительная к глютену целиакия</kwd><kwd>аутоиммунный гастрит</kwd><kwd>инфекция Helicobacter pylori</kwd></kwd-group></article-meta></front><body></body><back><ref-list><ref id="B1"><label>1.</label><mixed-citation>Новик А.А., Богданов А.Н. 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